<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE330nnn/GSE330217/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE330217</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Targeting the Disulfidptosis Key Gene SLC5A6 Inhibits HCC Proliferation and Improves the Immune Microenvironment</name><description>The sequencing samples comprised two human hepatocellular carcinoma cell lines, QGY-7701 and SMMC-7721. Each cell line was subjected to two distinct conditions to construct an in vitro disulfidptosis model: a Control group maintained in glucose-containing complete medium, and a Model group transitioned to glucose-free medium to induce glucose starvation. Two biological replicates were prepared for each condition, yielding a total of 8 samples (4 for QGY-7701 and 4 for SMMC-7721) for subsequent RNA extraction and high-throughput transcriptomic sequencing.</description><dates><publication>2026/09/02</publication></dates><accession>GSE330217</accession><cross_references><GSM>GSM9721428</GSM><GSM>GSM9721429</GSM><GSM>GSM9721426</GSM><GSM>GSM9721427</GSM><GSM>GSM9721425</GSM><GSM>GSM9721431</GSM><GSM>GSM9721432</GSM><GSM>GSM9721430</GSM><GPL>24676</GPL><GSE>330217</GSE><taxon>Homo sapiens</taxon><PMID>[42666251]</PMID></cross_references></HashMap>