<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE330nnn/GSE330569/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE330569</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic data of the placenta from post-term mice caused by the absence of steroid hormone receptor coactivators</name><description>Parturition depends on precise communication between the mother and fetus.Our previous study demonstrated that wild-type mice carrying fetuses doubly deficient in steroid receptor coactivators (Src)-1 and Src-2 (Src-1/-2d/d) manifested a delay in parturition of 1–2 days, caused by a deficiency in surfactant protein A (SP-A) and platelet-activating factor (PAF) production by the fetal lungs. To investigate whether placental dysfunction contributes to this phenotype and identify placenta-derived parturition signals, the current study investigated alterations in gene expression in the placentas of Src-1/-2 double-knockout (dko) fetuses.</description><dates><publication>2026/05/14</publication></dates><accession>GSE330569</accession><cross_references><GSM>GSM9728930</GSM><GSM>GSM9728932</GSM><GSM>GSM9728921</GSM><GSM>GSM9728931</GSM><GSM>GSM9728920</GSM><GSM>GSM9728934</GSM><GSM>GSM9728923</GSM><GSM>GSM9728933</GSM><GSM>GSM9728922</GSM><GSM>GSM9728925</GSM><GSM>GSM9728935</GSM><GSM>GSM9728924</GSM><GSM>GSM9728927</GSM><GSM>GSM9728926</GSM><GSM>GSM9728929</GSM><GSM>GSM9728928</GSM><GPL>24247</GPL><GSE>330569</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>