GEOapplication/xmlftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE331nnn/GSE331327/primary200OKTranscriptomicsMus musculusExpression profiling by high throughput sequencinghttps://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE331327GEOGSEfalseLoss of Foxf1 in lung pericytes increases the severity of pulmonary fibrosisMultiple cell types have been implicated in pathogenesis of pulmonary fibrosis (PF) with pericytes emerging as a new focus due to their role in promoting fibrotic remodeling. Fibrotic environment changes normal pericyte functions, but transcriptional programs regulating pericyte transition towards fibrotic state remain unclear. Utilizing scRNA-seq of mouse genetic models, we identified a unique cluster of fibrosis-associated pericytes.FOXF1 was identified as one of the transcription factors decreased in fibrotic pericytes.2026/08/19GSE331327GSM9791957GSM974402534328331327Mus musculus[42373667]