<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE332nnn/GSE332759/</Other></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE332759</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Intracellular complement factor H protects neurons during CNS inflammation [bulkRNA-seq]</name><description>Complement factor H (CFH) has been implicated in neuroprotection during inflammatory neurodegeneration, but its neuron-intrinsic functions remain poorly understood. Here, we performed bulk RNA sequencing of primary neuronal cultures from Cfh conditional knockout (Cfh-cKO) and control mice under basal conditions or following glutamate-induced oxidative stress. This dataset was generated to characterize transcriptional changes associated with neuronal Cfh deficiency and to identify pathways regulated by intracellular CFH during neuronal stress responses.</description><dates><publication>2026/07/28</publication></dates><accession>GSE332759</accession><cross_references><GSM>GSM9753017</GSM><GSM>GSM9753006</GSM><GSM>GSM9753016</GSM><GSM>GSM9753005</GSM><GSM>GSM9753019</GSM><GSM>GSM9753008</GSM><GSM>GSM9753018</GSM><GSM>GSM9753007</GSM><GSM>GSM9753009</GSM><GSM>GSM9753020</GSM><GSM>GSM9753011</GSM><GSM>GSM9753010</GSM><GSM>GSM9753013</GSM><GSM>GSM9753012</GSM><GSM>GSM9753015</GSM><GSM>GSM9753014</GSM><GPL>24247</GPL><GSE>332759</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>