<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE332nnn/GSE332971/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Homo sapiens</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE332971</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Hepatitis B virus protein X promotes survival and loss of hepatocyte identity in a differentiated human liver organoid system. [ATAC-Seq]</name><description>In this study, we leverage liver organoids derived from adult human livers to investigate HBx function in hepatocytes. Our findings demonstrate that HBx reshapes hepatocyte identity and apoptotic signaling to promote cell survival, establishing a relevant platform to study HBV persistence and liver tumorigenesis.</description><dates><publication>2026/09/01</publication></dates><accession>GSE332971</accession><cross_references><GSM>GSM9756311</GSM><GSM>GSM9756314</GSM><GSM>GSM9756312</GSM><GSM>GSM9756313</GSM><GPL>28038</GPL><GSE>332971</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>