<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE333nnn/GSE333507/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by array</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE333507</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Clonal expansion and phenotypic alterations of TCR Vβ3⁺ T cells in juvenile-onset recurrent respiratory papillomatosis: Implications for tumor-associated immunity and chemokine-mediated T-cell trafficking</name><description>Juvenile-onset recurrent respiratory papillomatosis (JORRP) is a rare, human papillomavirus (HPV)-driven pediatric disease characterized by recurring papillomas in the respiratory tract. The contribution of dysregulated T-cell immunity to disease severity remains poorly understood. This study included 97 JORRP patients and 124 controls</description><dates><publication>2026/06/01</publication></dates><accession>GSE333507</accession><cross_references><GSM>GSM9766557</GSM><GSM>GSM9766558</GSM><GSM>GSM9766559</GSM><GSM>GSM9766571</GSM><GSM>GSM9766550</GSM><GSM>GSM9766551</GSM><GSM>GSM9766552</GSM><GSM>GSM9766553</GSM><GSM>GSM9766554</GSM><GSM>GSM9766555</GSM><GSM>GSM9766556</GSM><GSM>GSM9766570</GSM><GSM>GSM9766568</GSM><GSM>GSM9766547</GSM><GSM>GSM9766569</GSM><GSM>GSM9766548</GSM><GSM>GSM9766549</GSM><GSM>GSM9766560</GSM><GSM>GSM9766561</GSM><GSM>GSM9766562</GSM><GSM>GSM9766563</GSM><GSM>GSM9766564</GSM><GSM>GSM9766565</GSM><GSM>GSM9766566</GSM><GSM>GSM9766567</GSM><GPL>21185</GPL><GSE>333507</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>