<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE333nnn/GSE333903/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type> Other</gds_type><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE333903</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Human metapneumovirus and mumps virus alter the m6A landscapes in THP-1-derived immature dendritic cells</name><description>N6-methyladenosine (m6A) is a widespread mRNA modification that regulates RNA metabolism and influences virus-induced immune responses. We performed MeRIP-seq to profile m6A landscapes in THP-1-derived immature dendritic cells (THP-iDCs) before and after infection with Human metapneumovirus (hMPV, NL/00/1) or mumps virus (MuV, JL2). Integration of m6A modifications on key innate immune transcripts with transcriptomic data, together with m6A levels on cellular and viral RNA, revealed a dual role of m6A in viral replication and host immune regulation. These datasets provide a resource for exploring epitranscriptomic regulation during hMPV and MuV infection.</description><dates><publication>2026/07/10</publication></dates><accession>GSE333903</accession><cross_references><GSM>GSM9777079</GSM><GSM>GSM9777078</GSM><GSM>GSM9777077</GSM><GSM>GSM9777087</GSM><GSM>GSM9777076</GSM><GSM>GSM9777086</GSM><GSM>GSM9777085</GSM><GSM>GSM9777084</GSM><GSM>GSM9777083</GSM><GSM>GSM9777082</GSM><GSM>GSM9777081</GSM><GSM>GSM9777080</GSM><GPL>29480</GPL><GSE>333903</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>