<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335386/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE335386</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Targeting FKBP5 Preserves Mitochondrial Homeostasis through the CaMKKβ/AMPK Pathway to Attenuate Sepsis-Induced Acute Lung Injury</name><description>Background: Sepsis-induced acute lung injury (ALI) remains a leading cause of mortality in critically ill patients, with endothelial barrier disruption and mitochondrial dysfunction serving as central pathological events. FK506-binding protein 5 (FKBP5) has been implicated in various inflammatory disorders, yet its specific role and underlying mechanism in septic ALI remain elusive.</description><dates><publication>2026/08/13</publication></dates><accession>GSE335386</accession><cross_references><GSM>GSM9812743</GSM><GSM>GSM9812742</GSM><GSM>GSM9812745</GSM><GSM>GSM9812744</GSM><GSM>GSM9812741</GSM><GSM>GSM9812747</GSM><GSM>GSM9812746</GSM><GSM>GSM9812749</GSM><GSM>GSM9812748</GSM><GPL>34284</GPL><GSE>335386</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>