{"database":"GEO","file_versions":[{"headers":{"Content-Type":["application/json"]},"body":{"files":{"Txt":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335495/suppl/filelist.txt"],"Raw":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335495/suppl/GSE335495_RAW.tar"],"Other":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335495/"]},"type":"primary"},"statusCode":"OK","statusCodeValue":200}],"scores":null,"additional":{"omics_type":["Genomics"],"species":["Homo sapiens"],"gds_type":["Genome variation profiling by genome tiling array"],"full_dataset_link":["https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE335495"],"repository":["GEO"],"entry_type":["GSE"],"additional_accession":[]},"is_claimable":false,"name":"Contribution of chromosomal aneuploidy and submicroscopic copy number variations (CNVs) in recurrent pregnancy loss (RPL)","description":"This study investigates the contribution of chromosomal aneuploidy and submicroscopic copy number variations (CNVs) in recurrent pregnancy loss (RPL). Genomic DNA from RPOC samples was analyzed using high-resolution Agilent aCGH arrays (180K). Several clinically relevant CNVs, including deletions and amplifications affecting immune-related and developmental genes, were identified. This suggests a genetic predisposition underlying some cases of RPL.","dates":{"publication":"2026/06/19"},"accession":"GSE335495","cross_references":{"GSM":["GSM9814650","GSM9814651","GSM9814647","GSM9814648","GSM9814656","GSM9814645","GSM9814646","GSM9814657","GSM9814654","GSM9814655","GSM9814652","GSM9814653","GSM9814649"],"GPL":["37117"],"GSE":["335495"],"taxon":["Homo sapiens"]}}