GEOapplication/xmlftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335568/suppl/filelist.txtftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335568/suppl/GSE335568_RAW.tarftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE335nnn/GSE335568/primaryOK200GenomicsHomo sapiensGenome binding/occupancy profiling by high throughput sequencinghttps://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE335568GEOGSEfalseThe FGF4–integrin β1 axis orchestrates diabetic wound regeneration by restoring directional collective motility.This dataset was generated to investigate the mechanisms underlying the pro-healing effects of Fibroblast Growth Factor 4 (FGF4) on diabetic wounds. We performed RNA sequencing on wound tissues from streptozotocin (STZ)-induced diabetic mice. The mice received daily topical applications of either recombinant FGF4 (rFGF4) or PBS vehicle control. Wound tissues were harvested on day 7 post-wounding for transcriptomic analysis.2026/06/18GSE335568GSM9815740GSM9815738GSM981573934284335568Homo sapiens