<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE337nnn/GSE337173/</Other></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE337173</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic profiling of GOLM1 silenced A549 lung adenocarcinoma cells</name><description>GOLM1 is a Golgi-resident protein implicated in multiple cancers, but its role in lung adenocarcinoma remains incompletely understood. RNA sequencing was performed on A549 lung adenocarcinoma cells following siRNA-mediated silencing of GOLM1 using two independent siRNAs and a non-targeting control. Differential gene expression analysis was conducted to identify molecular pathways regulated by GOLM1 depletion, including alterations in cell cycle regulation, DNA replication, lysosomal pathways, and mitochondrial-associated processes.</description><dates><publication>2026/07/04</publication></dates><accession>GSE337173</accession><cross_references><GSM>GSM9849887</GSM><GSM>GSM9849888</GSM><GSM>GSM9849885</GSM><GSM>GSM9849886</GSM><GSM>GSM9849889</GSM><GSM>GSM9849890</GSM><GSM>GSM9849891</GSM><GSM>GSM9849894</GSM><GSM>GSM9849895</GSM><GSM>GSM9849884</GSM><GSM>GSM9849892</GSM><GSM>GSM9849893</GSM><GPL>34284</GPL><GSE>337173</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>