<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE338nnn/GSE338324/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE338324</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Hemin-induced DEGs in Lung, Kidney of WT and hepatic SR-BI KO mice</name><description>Acute hemolysis can deplete haptoglobin and hemopexin, leaving toxic free heme insufficiently controlled. Here, the authors show that scavenger receptor BI (SR-BI) mediates hepatic uptake of free heme and is essential for protection against heme-induced injury and death. Here we examined gene expression post hemin injections</description><dates><publication>2026/07/15</publication></dates><accession>GSE338324</accession><cross_references><GSM>GSM9871377</GSM><GSM>GSM9871366</GSM><GSM>GSM9871365</GSM><GSM>GSM9871376</GSM><GSM>GSM9871379</GSM><GSM>GSM9871368</GSM><GSM>GSM9871367</GSM><GSM>GSM9871378</GSM><GSM>GSM9871369</GSM><GSM>GSM9871380</GSM><GSM>GSM9871382</GSM><GSM>GSM9871371</GSM><GSM>GSM9871370</GSM><GSM>GSM9871381</GSM><GSM>GSM9871373</GSM><GSM>GSM9871372</GSM><GSM>GSM9871375</GSM><GSM>GSM9871374</GSM><GPL>24247</GPL><GSE>338324</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>