<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Bed>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE341nnn/GSE341816/suppl/GSE341816_HBV_summits.bed.gz</Bed><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE341nnn/GSE341816/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Other</omics_type><species>Homo sapiens</species><gds_type>Other</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE341816</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>The pseudouridine epitranscroptome of PA-ψ-seq HepAD38 tet- cell line</name><description>Persistent HBV replication remodels epitranscriptomic modifications in hepatocytes. Pseudouridine (Ψ) is a prevalent RNA modification regulating RNA stability and translation, yet the landscape of pseudouridylation during HBV replication remains unclear. Herein, we applied Photo-Crosslinking-Assisted pseudouridine sequencing (PA-Ψ-seq) to profile the transcriptome-wide pseudouridine epitranscriptome in HepAD38 tet⁻ cells with active HBV replication.</description><dates><publication>2026/09/17</publication></dates><accession>GSE341816</accession><cross_references><GSM>GSM9917791</GSM><GSM>GSM9917792</GSM><GSM>GSM9917793</GSM><GPL>24676</GPL><GSE>341816</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>