<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE342nnn/GSE342961/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE342961</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic profiling (RNA-seq) of lung endothelial cells from wild-type (WT) and TET2KO mice following 28 days of Angiotensin II treatment.</name><description>Chronic hypertension and Angiotensin II infusion induce systemic vascular and endothelial dysfunction. To investigate cell-autonomous transcriptional changes resulting from endothelial-specific TET2 deficiency in the pulmonary microenvironment, we performed total RNA sequencing on lung endothelial cells isolated from wild-type (WT) and endothelial-specific TET2 knockout (TET2KO) mice after 28 days of Angiotensin II treatment.</description><dates><publication>2026/08/10</publication></dates><accession>GSE342961</accession><cross_references><GSM>GSM9944068</GSM><GSM>GSM9944067</GSM><GSM>GSM9944064</GSM><GSM>GSM9944066</GSM><GSM>GSM9944065</GSM><GPL>24247</GPL><GSE>342961</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>