<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE343nnn/GSE343228/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE343228</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>TGF-β1/c-Myc/PKM2 Axis in Endometrial Decidualization Impairment in Adenomyosis: Mechanistic Insights and Therapeutic Implications</name><description>Following the addition of exogenous transforming growth factor-beta 1 (TGF-β1) to human endometrial stromal cells, a phenotype mimicking the increased TGF-β1 expression observed in the endometrium of patients with adenomyosis was established. RNA sequencing was employed to identify differential gene expression between the treated and control groups, with the aim of elucidating the mechanisms underlying aberrant decidualization of endometrial stromal cells.</description><dates><publication>2026/08/17</publication></dates><accession>GSE343228</accession><cross_references><GSM>GSM9948007</GSM><GSM>GSM9948006</GSM><GSM>GSM9948005</GSM><GSM>GSM9948004</GSM><GSM>GSM9948003</GSM><GSM>GSM9948002</GSM><GPL>34284</GPL><GSE>343228</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>