{"database":"GEO","file_versions":[{"headers":{"Content-Type":["application/json"]},"body":{"files":{"Other":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE343nnn/GSE343411/"]},"type":"primary"},"statusCode":"OK","statusCodeValue":200}],"scores":null,"additional":{"omics_type":["Genomics"],"species":["Homo sapiens"],"gds_type":["Genome binding/occupancy profiling by high throughput sequencing"],"full_dataset_link":["https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE343411"],"repository":["GEO"],"entry_type":["GSE"],"additional_accession":[]},"is_claimable":false,"name":"Integrated chromatin accessibility and transcriptomic profiling reveals inflammation-associated endothelial remodeling in capillary malformation iECs","description":"Integrated bulk ATAC-seq and RNA-seq profiling of induced endothelial cells (iECs) derived from capillary malformation (CM) lesion-derived and control induced pluripotent stem cells (iPSCs). Differential chromatin accessibility, gene expression, transcription factor activity, and candidate peak-to-gene links were analyzed to identify regulatory programs associated with endothelial differentiation and remodeling in CM.","dates":{"publication":"2026/09/30"},"accession":"GSE343411","cross_references":{"GSM":["GSM9950937","GSM9950936","GSM9950939","GSM9950938","GSM9950935"],"GPL":["24676"],"GSE":["343411"],"taxon":["Homo sapiens"],"PMID":["[42788004]"]}}