<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE343nnn/GSE343411/</Other></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Homo sapiens</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE343411</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Integrated chromatin accessibility and transcriptomic profiling reveals inflammation-associated endothelial remodeling in capillary malformation iECs</name><description>Integrated bulk ATAC-seq and RNA-seq profiling of induced endothelial cells (iECs) derived from capillary malformation (CM) lesion-derived and control induced pluripotent stem cells (iPSCs). Differential chromatin accessibility, gene expression, transcription factor activity, and candidate peak-to-gene links were analyzed to identify regulatory programs associated with endothelial differentiation and remodeling in CM.</description><dates><publication>2026/09/30</publication></dates><accession>GSE343411</accession><cross_references><GSM>GSM9950937</GSM><GSM>GSM9950936</GSM><GSM>GSM9950939</GSM><GSM>GSM9950938</GSM><GSM>GSM9950935</GSM><GPL>24676</GPL><GSE>343411</GSE><taxon>Homo sapiens</taxon><PMID>[42788004]</PMID></cross_references></HashMap>