<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE343nnn/GSE343805/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Homo sapiens</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE343805</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>KLF2-dependent chromatin accessibility changes in hepatic stellate cells</name><description>Krüppel-like factor 2 (KLF2) is a transcription factor involved in regulating cellular homeostasis and diverse signaling responses. To investigate KLF2-associated changes in chromatin accessibility in human hepatic stellate cells, we performed ATAC-seq on LX-2 cells stably expressing KLF2 or control vector. Genome-wide chromatin accessibility profiles were compared between the two groups to identify KLF2-dependent changes. This dataset provides a resource for exploring the potential epigenomic mechanisms underlying KLF2-mediated regulation of hepatic stellate cell function.</description><dates><publication>2026/08/21</publication></dates><accession>GSE343805</accession><cross_references><GSM>GSM9963034</GSM><GSM>GSM9963033</GSM><GSM>GSM9963036</GSM><GSM>GSM9963035</GSM><GPL>24676</GPL><GSE>343805</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>