{"database":"GEO","file_versions":[{"headers":{"Content-Type":["application/json"]},"body":{"files":{"Other":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE345nnn/GSE345466/"]},"type":"primary"},"statusCode":"OK","statusCodeValue":200}],"scores":null,"additional":{"omics_type":["Transcriptomics"],"species":["Homo sapiens"],"gds_type":["Expression profiling by high throughput sequencing"],"full_dataset_link":["https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE345466"],"repository":["GEO"],"entry_type":["GSE"],"additional_accession":[]},"is_claimable":false,"name":"Developmental Arrest Associated with Altered Cerebellar Metabolism in Sudden Infant Death Syndrome","description":"Sudden infant death syndrome (SIDS) may involve impaired cerebellar contributions to autonomic and arousal responses during hypoxic stress. We performed bulk RNA sequencing of flash-frozen postmortem human cerebellar cortex from SIDS infants and age-matched non-SIDS controls to define transcriptional alterations associated with SIDS. Differential-expression and pathway-enrichment analyses were used to evaluate neuroinflammatory, neurotransmitter, and metabolic programs, and transcriptomic findings were compared at the pathway level with independently generated targeted metabolomics and quantitative histology data.","dates":{"publication":"2026/08/30"},"accession":"GSE345466","cross_references":{"GSM":["GSM10005134","GSM10005145","GSM10005144","GSM10005133","GSM10005136","GSM10005135","GSM10005146","GSM10005141","GSM10005130","GSM10005140","GSM10005132","GSM10005143","GSM10005142","GSM10005131","GSM10005138","GSM10005137","GSM10005129","GSM10005139"],"GPL":["24676"],"GSE":["345466"],"taxon":["Homo sapiens"]}}