<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE348nnn/GSE348238/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Homo sapiens</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE348238</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>SMARCA4 CUT&amp;Tag profiling in parental and ARID1A-knockdown BxPC3 pancreatic cancer clones</name><description>This Series profiles SMARCA4 (BRG1) chromatin occupancy by CUT&amp;Tag in parental BxPC3 pancreatic ductal adenocarcinoma cells and selected control or ARID1A-knockdown clonal populations. The data were generated to examine whether residual SMARCA4 preferentially occupies regulatory regions associated with increased and variable chromatin accessibility after ARID1A loss. Drosophila spike-in nuclei were included for quantitative normalization.</description><dates><publication>2026/09/22</publication></dates><accession>GSE348238</accession><cross_references><GSM>GSM10068926</GSM><GSM>GSM10068925</GSM><GSM>GSM10068929</GSM><GSM>GSM10068928</GSM><GSM>GSM10068927</GSM><GPL>21697</GPL><GSE>348238</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>