<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE87nnn/GSE87533/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Mus musculus</species><gds_type>Non-coding RNA profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE87533</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Small RNA Sequencing of mouse amygdala after knockdown of miR-15a</name><description>MicroRNAs are important regulators of gene expression and associated with stress-related psychiatric disorders. We report that exposing mice to chronic stress led to a specific increase in microRNA-15a levels in the amygdala-Ago2 complex, and a concomitant reduction in the levels of its predicted target, FKBP51, which is implicated in stress-related psychiatric disorders. Reciprocally, mice expressing reduced levels of amygdalar microRNA-15a following exposure to chronic stress exhibited increased anxiety-like behaviors. Here, we performed small RNA Sequencing of mouse basolateral amygdala after miR15a knockdown using injection of a miR-15a sponge virus or control sponge virus.</description><dates><publication>2016/10/01</publication></dates><accession>GSE87533</accession><cross_references><GSM>GSM2333473</GSM><GSM>GSM2333474</GSM><GSM>GSM2333471</GSM><GSM>GSM2333472</GSM><GSM>GSM2333470</GSM><GSM>GSM2333468</GSM><GSM>GSM2333469</GSM><GSM>GSM2333466</GSM><GSM>GSM2333467</GSM><GSM>GSM2333465</GSM><GPL>16417</GPL><SRA>SRP090706</SRA><GSE>87533</GSE><taxon>Mus musculus</taxon><PMID>[27829158]</PMID></cross_references></HashMap>