{"database":"iProX","file_versions":[],"scores":null,"additional":{"omics_type":["Proteomics"],"submitter":["Bin Liu"],"species":["Homo Sapiens"],"full_dataset_link":["http://www.iprox.org/page/project.html?id=IPX0016139000"],"submitter_email":["binliu@jlu.edu.cn"],"submitter_affiliation":["Jilin University"],"sample_protocol":[""],"repository":["iProX"],"data_protocol":[""],"additional_accession":[]},"is_claimable":false,"name":"Laminar shear stress inhibits endothelial-mesenchymal transition and improves atherosclerosis through SRXN1","description":"Atherosclerosis (AS) is one of the main causes of cardiovascular diseases, and its complex pathophysiological mechanism involves the interaction of multiple factors.    Laminar shear stress (LSS) plays a key role in the regulation of  AS processes, but its specific mechanism is still unclear.     In this study, we analyzed the proteome data of static culture and LSS-treated human aortic endothelial cells (HAECs), to explore the role and potential mechanism of LSS in AS.","dates":{"publication":"Thu Mar 19 00:00:00 GMT 2026"},"accession":"PXD075827","cross_references":{"TAXONOMY":["9606"]}}