<HashMap><database>iProX</database><scores/><additional><omics_type>Proteomics</omics_type><submitter>Bin Liu</submitter><species>Homo Sapiens</species><full_dataset_link>http://www.iprox.org/page/project.html?id=IPX0016139000</full_dataset_link><submitter_email>binliu@jlu.edu.cn</submitter_email><submitter_affiliation>Jilin University</submitter_affiliation><sample_protocol></sample_protocol><repository>iProX</repository><data_protocol></data_protocol></additional><is_claimable>false</is_claimable><name>Laminar shear stress inhibits endothelial-mesenchymal transition and improves atherosclerosis through SRXN1</name><description>Atherosclerosis (AS) is one of the main causes of cardiovascular diseases, and its complex pathophysiological mechanism involves the interaction of multiple factors.    Laminar shear stress (LSS) plays a key role in the regulation of  AS processes, but its specific mechanism is still unclear.     In this study, we analyzed the proteome data of static culture and LSS-treated human aortic endothelial cells (HAECs), to explore the role and potential mechanism of LSS in AS.</description><dates><publication>Thu Mar 19 00:00:00 GMT 2026</publication></dates><accession>PXD075827</accession><cross_references><TAXONOMY>9606</TAXONOMY></cross_references></HashMap>