<HashMap><database>iProX</database><scores/><additional><omics_type>Proteomics</omics_type><submitter>Xianwei Wang</submitter><species>Homo Sapiens</species><full_dataset_link>http://www.iprox.org/page/project.html?id=IPX0016598000</full_dataset_link><submitter_email>Wangxianwei1116@126.com</submitter_email><submitter_affiliation>Henan Key Laboratory of Medical Tissue Regeneration</submitter_affiliation><sample_protocol></sample_protocol><repository>iProX</repository><data_protocol></data_protocol></additional><is_claimable>false</is_claimable><name>Proteomics of AC-16 cells treated with DOX</name><description>Doxorubicin (DOX) is a potent chemotherapeutic agent, but its clinical use is limited by dose-dependent cardiotoxicity. To elucidate the molecular mechanisms underlying DOX-induced cardiomyopathy, we performed quantitative proteomic analysis on DOX-treated human AC-16 cardiomyocytes. The treatment group comprised AC-16 cells exposed to 0.2 µM DOX for 48 hours, with solvent-treated cells serving as the control.</description><dates><publication>Mon Apr 13 00:00:00 BST 2026</publication></dates><accession>PXD077062</accession><cross_references><TAXONOMY>9606</TAXONOMY></cross_references></HashMap>