<HashMap><database>MassIVE</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://massive-ftp.ucsd.edu/v02/MSV000083349/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores><citationCount>0</citationCount><reanalysisCount>0</reanalysisCount><viewCount>0</viewCount><searchCount>0</searchCount></scores><additional><submitter>Anne-Claude Gingras</submitter><full_dataset_link>https://massive.ucsd.edu/ProteoSAFe/dataset.jsp?task=092651d0b0dd40c39613bbc5efdeb606</full_dataset_link><submitter_email>gingras@lunenfeld.ca</submitter_email><sample_protocol></sample_protocol><repository>MassIVE</repository><file_size>42</file_size><ptm_modification>MOD:00685 - "A protein modification that effectively converts an L-glutamine residue to L-glutamic acid."</ptm_modification><ptm_modification>MOD:00684 - "A protein modification that effectively converts an L-asparagine residue to L-aspartic acid."</ptm_modification><ptm_modification>MOD:00719 - "A protein modification that oxygenates an L-methionine residue to one of the diastereomeric L-methionine sulfoxide residues."</ptm_modification><data_protocol></data_protocol><omics_type>Proteomics</omics_type><instrument_platform>TripleTOF 6600</instrument_platform><species>Mus Musculus (ncbitaxon:10090)</species><submitter_affiliation>LTRI</submitter_affiliation><description_synonyms>protein translation, Cognitive Function, Bru, protein anabolism, dTAF[[II]]230, TAF[[II]]250, d230, P220, Disorders, protein biosynthetic process, Data Set, Raw, Kanner's Syndrome, TAF200, l(3)84Ab, dTAFII250, BG:DS00004.13, Infantile, TAFII-250, TAF250/230, EfW1, not genetically inherited, Cell, dTAF230, Concept, cognitive function, dmTAF[[II]]230, Insights, TAFII250, Role Concept, dmTAF1, Roles, Taf230, p230, Infantile Autism, PARK18, Role, Kanners Syndrome, protein formation, TAF[[II]]250/230, Concepts, TFIID, protein biosynthesis, Del(8)44H, Early Infantile, Early, susceptiblity to, TAF250, Svc, Taf[[II]]250, Kanner Syndrome, Taf200, Col4a-1, dTAF[[II]]250, TAF[[II]]230, TFIID TAF250, cel, cell, Early Infantile Autism, Autistic, Function, Taf1p, TAF[II]250, E030015G23Rik, EIF4GI, CG17603, EIF4F, Cognitive, TAF[[II]], EIF4G, EIF-4G1, Cognitive Functions, dTAF250, Cognitions, DmelCG17603, Taf250, protein synthesis, eIF4GI, Functions, SR3-5, Disorder, Manuscripts, Role Concepts, autism, Insight., Autism, associated, TAF, TAF230, TAF1</description_synonyms><name_synonyms>P220, E030015G23Rik, EIF4GI, eIF4GI, EIF4F, EIF4G, EIF-4G1, PARK18.</name_synonyms><citation_count>0</citation_count><additional_accession>PXD012421</additional_accession></additional><is_claimable>false</is_claimable><name>Gonatopoulos_eIF4G1_microexon_BioID</name><description>This dataset consists of 7 raw MS files and associated peak lists and result files, acquired on a TripleTOF 6600 spectrometer (AB SCIEX, Concord, Ontario, Canada) operated in Data Dependent Acquisition mode.  Cell pellets were generated by Thomas Gonatopoulos with BioID samples prepared and analyzed by Jonathan Roth. These files are associated with a manuscript by Thomas Gonatopoulos-Pournatzis et al that describes the functional role of an autism regulated microexon in eIF4G1 and its role in regulating translation and cognitive function.</description><dates><publication>Thu Jan 17 22:31:00 GMT 2019</publication></dates><accession>MSV000083349</accession><cross_references/></HashMap>