<HashMap><database>MetabolomicsWorkbench</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>http://www.metabolomicsworkbench.org/rest/study/study_id/ST001185/mwtab</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><technology_type>Mass Spectrometry</technology_type><technology_type>ESI</technology_type><disease>Fatty Liver Disease</disease><full_dataset_link>http://www.metabolomicsworkbench.org/data/DRCCMetadata.php?Mode=Study&amp;StudyID=ST001185</full_dataset_link><study_factor>Source:Healthy Control Liver Tissue</study_factor><study_factor>Source:Negative Control Empty Scaffold</study_factor><study_factor>Source:NASH Liver Tissue from Patient</study_factor><study_factor>Source:Bioengineered human iPS-derived fatty liver tissue-iKD-SIRT1</study_factor><tissue>Liver</tissue><sample_protocol>Reversed phase POSITIVE ION MODE. Reversed phase NEGATIVE ION MODE. Reversed phase UNSPECIFIED ION MODE.</sample_protocol><repository>MetabolomicsWorkbench</repository><data_protocol></data_protocol><omics_type>Metabolomics</omics_type><species>Homo Sapiens</species><submitter_affiliation>University of Pittsburgh</submitter_affiliation><metabolite_name>Glutamic acid</metabolite_name><metabolite_name>Hexose</metabolite_name><metabolite_name>Serine</metabolite_name><metabolite_name>Pyruvic acid</metabolite_name><metabolite_name>Leucine</metabolite_name><metabolite_name>Itaconic acid</metabolite_name><metabolite_name>Fructose-1,6-biphosphate</metabolite_name><metabolite_name>Histidine</metabolite_name><metabolite_name>Arginine</metabolite_name><metabolite_name>Aspartic acid</metabolite_name><metabolite_name>Tyrosine</metabolite_name><metabolite_name>Creatinine</metabolite_name><metabolite_name>Adenosine</metabolite_name><metabolite_name>Alanine</metabolite_name><metabolite_name>Phosphoenolpyruvic acid</metabolite_name><metabolite_name>Methionine</metabolite_name><metabolite_name>Oxoglutaric acid</metabolite_name><metabolite_name>Tryptophan</metabolite_name><metabolite_name>CAR(2:0)</metabolite_name><metabolite_name>Glucose-6-phosphate</metabolite_name><metabolite_name>Citric acid</metabolite_name><metabolite_name>Fumaric acid</metabolite_name><metabolite_name>Valine</metabolite_name><metabolite_name>Lysine</metabolite_name><metabolite_name>Succinic acid</metabolite_name><metabolite_name>Cis-Aconitic acid</metabolite_name><metabolite_name>Lactic acid</metabolite_name><metabolite_name>Ornithine</metabolite_name><metabolite_name>Glutamine</metabolite_name><name_synonyms>Steatohepatitides, fatty change of liver, AA673258, Sir2alpha, Liver, human being, Man (Taxonomy), Visceral, Liver Steatoses, fatty liver, Fatty infiltration of liver, Liver Steatosis, Modern, familial, Tissue, Sir2, Fatty liver, sirtuin, Steatohepatitis, SIR2L1, Steatosis of Liver, steatosis of liver, human, genetic, Human, sir2l1, Steatoses, Visceral Steatoses, Sir2a, Homo sapiens, Silent Mating Type Information Regulation 2 Homolog 1., Visceral Steatosis, Modern Man, Steatosis, inherited genetic, hepatic lipidosis, simple tissue, Fatty, constitutitional genetic, hereditary, Sirt1, Man, Liver steatosis</name_synonyms><sample_synonyms>iones., backward, Ionen, ion, Ion, ions, reversed</sample_synonyms></additional><is_claimable>false</is_claimable><name>Genetic and metabolic characterization of bioengineered human fatty liver tissue with modified SIRT1 expression</name><description/><dates><publication></publication></dates><accession>ST001185</accession><cross_references><TAXONOMY>9606</TAXONOMY></cross_references></HashMap>