<HashMap><database>MetabolomicsWorkbench</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>http://www.metabolomicsworkbench.org/rest/study/study_id/ST001186/mwtab</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Metabolomics</omics_type><technology_type>Mass Spectrometry</technology_type><technology_type>ESI</technology_type><disease>Huntingtons Disease</disease><species>Mus Musculus</species><full_dataset_link>http://www.metabolomicsworkbench.org/data/DRCCMetadata.php?Mode=Study&amp;StudyID=ST001186</full_dataset_link><study_factor>Time:3 | Treatment:Dimethyl fumarate</study_factor><study_factor>Time:1 | Treatment:4-Deoxypyridoxine</study_factor><study_factor>Time:1 | Treatment:Sodium Butyrate</study_factor><study_factor>Time:2 | Treatment:Trichostatin A</study_factor><study_factor>Time:3 | Treatment:Cyproheptadine</study_factor><study_factor>Time:3 | Treatment:Loxapine</study_factor><study_factor>Time:2 | Treatment:Q111SST_Control</study_factor><study_factor>Time:1 | Treatment:Selisistat</study_factor><study_factor>Time:3 | Treatment:Trichostatin A</study_factor><study_factor>Time:2 | Treatment:Cyproheptadine</study_factor><study_factor>Time:1 | Treatment:Clioquinol</study_factor><study_factor>Time:1 | Treatment:Q7SST_Control</study_factor><study_factor>Time:1 | Treatment:Haloperidol</study_factor><study_factor>Time:3 | Treatment:Cysteamine</study_factor><study_factor>Time:2 | Treatment:4-Deoxypyridoxine</study_factor><study_factor>Time:1 | Treatment:Cyproheptadine</study_factor><study_factor>Time:2 | Treatment:Fingolimod phosphate</study_factor><study_factor>Time:2 | Treatment:Pizotifen</study_factor><study_factor>Time:2 | Treatment:Loxapine</study_factor><study_factor>Time:1 | Treatment:Trichostatin A</study_factor><study_factor>Time:3 | Treatment:Diacylglycerol kinase inhibitor II</study_factor><study_factor>Time:2 | Treatment:Sodium Butyrate</study_factor><study_factor>Time:1 | Treatment:Pizotifen</study_factor><study_factor>Time:3 | Treatment:Haloperidol</study_factor><study_factor>Time:3 | Treatment:Clioquinol</study_factor><study_factor>Time:2 | Treatment:Cysteamine</study_factor><study_factor>Time:2 | Treatment:Diacylglycerol kinase inhibitor II</study_factor><study_factor>Time:3 | Treatment:Meclizine</study_factor><study_factor>Time:1 | Treatment:Dimethyl fumarate</study_factor><study_factor>Time:1 | Treatment:Meclizine</study_factor><study_factor>Time:1 | Treatment:Loxapine</study_factor><study_factor>Time:3 | Treatment:Selisistat</study_factor><study_factor>Time:3 | Treatment:Pizotifen</study_factor><study_factor>Time:1 | Treatment:Nortriptyline</study_factor><study_factor>Time:2 | Treatment:Meclizine</study_factor><study_factor>Time:1 | Treatment:Q111SST_Control</study_factor><study_factor>Time:3 | Treatment:4-Deoxypyridoxine</study_factor><study_factor>Time:2 | Treatment:Nicotinamide</study_factor><study_factor>Time:3 | Treatment:Sodium Butyrate</study_factor><study_factor>Time:1 | Treatment:Cysteamine</study_factor><study_factor>Time:2 | Treatment:Nortriptyline</study_factor><study_factor>Time:2 | Treatment:Q7SST_Control</study_factor><study_factor>Time:3 | Treatment:Fingolimod phosphate</study_factor><study_factor>Time:3 | Treatment:Nicotinamide</study_factor><study_factor>Time:1 | Treatment:Diacylglycerol kinase inhibitor II</study_factor><study_factor>Time:1 | Treatment:Fingolimod phosphate</study_factor><study_factor>Time:2 | Treatment:Dimethyl fumarate</study_factor><study_factor>Time:2 | Treatment:Selisistat</study_factor><study_factor>Time:3 | Treatment:Nortriptyline</study_factor><study_factor>Time:2 | Treatment:Clioquinol</study_factor><study_factor>Time:3 | Treatment:Q111SST_Control</study_factor><study_factor>Time:1 | Treatment:Nicotinamide</study_factor><study_factor>Time:3 | Treatment:Q7SST_Control</study_factor><study_factor>Time:2 | Treatment:Haloperidol</study_factor><tissue>Cultured Cells</tissue><submitter_affiliation>Massachusetts Institute of Technology</submitter_affiliation><sample_protocol>Normal phase POSITIVE ION MODE. Normal phase NEGATIVE ION MODE. HILIC POSITIVE ION MODE. HILIC NEGATIVE ION MODE.</sample_protocol><repository>MetabolomicsWorkbench</repository><data_protocol></data_protocol><name_synonyms>preventive measures, Controlling, preventive therapy, reference sample, control, prophylaxis, Metabonomic, Cell., Metabonomics, Metabolomic, prevention and control, Cell, Controlled, prevention</name_synonyms><sample_synonyms>average, average.</sample_synonyms></additional><is_claimable>false</is_claimable><name>Untargeted metabolomics on control and compound-treated STHdhQ111 cells and control STHdhQ7 cells</name><description/><dates><publication></publication></dates><accession>ST001186</accession><cross_references><TAXONOMY>10090</TAXONOMY></cross_references></HashMap>