ENAapplication/xmlftp.sra.ebi.ac.uk/vol1/fastq/SRR303/002/SRR3033172/SRR3033172.fastq.gzftp.sra.ebi.ac.uk/vol1/fastq/SRR303/001/SRR3033171/SRR3033171.fastq.gzftp.sra.ebi.ac.uk/vol1/fastq/SRR303/000/SRR3033170/SRR3033170.fastq.gzprimaryOK2000000GenomicsDepartment of Medicine, UW-Madisonhttps://www.ebi.ac.uk/ena/browser/view/PRJNA306509Homo sapiensWe report the application of ChIP Seq to study the Epstein Barr Virus Nuclear Antigen EBNA3A, EBNA3B, EBNA3C, an essential transcriptional regulator involved in the transformation of Resting B Lymphocytes to the immortalized Lymphoblast Cell Lines. Overall design: Examination of EBNA3A, EBNA3B and EBNA3C protein genome binding in LCLs.ENASu(H), RBPjk, RBPJK, distinct, ligand, Proteins, AI843960, Gene, CBF1, proteins, Igkrsbp., XSu(H), Protein Gene Products, KBF2, Rbpsuh, Gene Proteins, Igkjrb, SUH, RBP-J kappa, Protein, xsu(h)1, Gene Products, AOS3, IGKJRB, RBPSUH, IGKJRB1, csl, RBP-Jhuman being, human., man00.00.00.00.00.0falseHomo sapiensEBNA3 proteins regulate EBNA2 binding to distinct RBPJ genomic sites2022-05-122016-01-13PRJNA306509GSE76166267192689606