ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/DRR255/DRR255657/DRR255657.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/DRR255/DRR255660/DRR255660.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/DRR255/DRR255658/DRR255658.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/DRR255/DRR255661/DRR255661.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/DRR255/DRR255659/DRR255659.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/DRR255/DRR255656/DRR255656.fastq.gzprimaryOK200GenomicsNational Institute Neuroscience, National Center of Neurology and Psychiatryhttps://www.ebi.ac.uk/ena/browser/view/PRJDB10791Mus musculusDifferentiation of oligodendrocyte is an important process during remyelination. Loss-of-function genomic screen identified dimethylarginine dimethylaminohydrolase 1 (DDAH1) as a novel regulator of oligodendrocyte differentiation.To investigate how DDAH1 regulate oligodendrocyte differentiation, RNA-seq analysis was performed to reveal the change of gene expression by DDAH1 knockdown in oligodendrocytes.ENADDAH, 2510015N06Rik, 2410006N07Rik, AI987801., HEL-S-16, AW050362Mus musculus, Laboratory Mice., House, Mus, Laboratory, Swiss, Mus domesticus, mouse, Mus musculus domesticus, Swiss Mouse, mouse <Mus musculus>, Mouse, House Mice, Swiss Mice, house mouse, Mice, Laboratory Mouse, House Mouse, mice C57BL/6xCBA/CaJ hybrid, domesticus, Mus muscarisfalseMus musculusExpression profiles in DDAH1 knockdown cultured oligodendrocytes2025-09-242020-12-09PRJDB1079110090