ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/007/ERR6090137/ERR6090137.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/009/ERR6090139/ERR6090139.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/008/ERR6090138/ERR6090138.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/003/ERR6090133/ERR6090133.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/004/ERR6090134/ERR6090134.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/005/ERR6090135/ERR6090135.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/ERR609/006/ERR6090136/ERR6090136.fastq.gzprimaryOK200GenomicsIMBUQEuropean Bioinformatics Institutehttps://www.ebi.ac.uk/ena/browser/view/PRJEB45796ArrayExpressWe investigated differences in the chromatin-binding profile of SOX18 (mouse) and its dominant-negative isoform SOX18RaOp, and how SOX18RaOp alters the binding location of SOX18. HeLa cells were transfected to generate 3 different conditions: 1. myc-SOX18, 2. myc-SOX18RaOp and 3. myc-SOX18 with untagged-SOX18RaOp. Each condition was performed in duplicate (6 samples total). An anti-Myc antibody was used to perform the pull-down. Background (input) was assessed by pooling cells transfected with each condition and sequencing without immunoprecipitation.ENAfalseChIP-seq of wild-type SOX18 and a dominant-negative variant (SOX18RaOp) in HeLa cellsChIP-seq of wild-type SOX18 and a dominant-negative variant (SOX18RaOp) in HeLa cells2021-08-302021-08-30PRJEB45796