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These findings suggest that loss of Dscc1 in mammalian cells should be incompatible with embryonic development, yet we have generated a Dscc1-null mouse. Primary cells derived from Dscc1-null mice exhibit the replication defect but show normal cohesion. To ascertain whether cohesion is rescued by mutation of a ‘bypass’ gene, we wish to exome sequence a number of Dscc1-null mice.ENAMus musculus, House, Mus, Laboratory, Swiss, mice, Mus domesticus, mouse, Mus musculus domesticus, Swiss Mouse, Exomes, Mouse, House Mice, Swiss Mice, house mouse, whole exome., Mice, Laboratory Mouse, House Mouse, domesticus, Laboratory MiceMus musculus, House, Mus, Laboratory, Swiss, mice, Mus domesticus, mouse, Mus musculus domesticus, Swiss Mouse, Exomes, Mouse, House Mice, Swiss Mice, house mouse, whole exome., Mice, Laboratory Mouse, House Mouse, domesticus, Laboratory MicefalseDscc1_mouse_exome_sequencingDscc1_mouse_exome_sequencing2016-05-202014-05-27PRJEB5346