ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR276/059/SRR27665259/SRR27665259_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR276/062/SRR27665262/SRR27665262_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR276/061/SRR27665261/SRR27665261_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR276/060/SRR27665260/SRR27665260_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR276/063/SRR27665263/SRR27665263_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR276/058/SRR27665258/SRR27665258_1.fastq.gzprimaryOK200GenomicsGenome Institute of Singaporehttps://www.ebi.ac.uk/ena/browser/view/PRJNA1066997We have identified PARP4 as a novel modulator of lung adenocarcinoma from a tumor sequencing study on what is currently the largest East Asian lung adenocarcinoma cohort (PMID: 32015526). We identified hnRNPM as a novel interaction partner of PARP4 and wanted to determine the role of hnRNPM and PARP4 in regulating splicing. We thus performed long-read Nanopore sequencing to study this.ENAfalseNanopore direct cDNA sequencing of hnRNPM and PARP4 knockdown lung cancer cells2024-04-042024-04-04PRJNA1066997