ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR288/029/SRR28818129/SRR28818129.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR288/028/SRR28818128/SRR28818128.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR288/032/SRR28818132/SRR28818132.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR288/030/SRR28818130/SRR28818130.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR288/031/SRR28818131/SRR28818131.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR288/027/SRR28818127/SRR28818127.fastq.gzprimaryOK200GenomicsErcan, Biology, New York Universityhttps://www.ebi.ac.uk/ena/browser/view/PRJNA1105103Caenorhabditis elegansIn embryos, DCC binding across the length of the ~17.7 Mb X chromosome initiates at a set of recruitment elements on the X (rex). Individual rex sites collectively contribute to recruitment and repression, thus deletion of one or few rex sites causes subtle changes, as measured by mRNA-seq in mixed-stage embryos. We previously constructed a strain deleting rex-1, located ~6 kb downstream of dpy-23 (https://doi.org/10.7554/eLife.23645). Here we performed mRNA-seq in rex-1 deletion embryos. Overall design: mRNA-seq in controls and rex-1 deletion embryos.ENAZfp-42, mRNA-seq, determination, Rex1, REX-1, chemical analysis, assay, Zinc finger protein 754, mREX-1, Rex-1, DEL., hREX-1, Reduced expression protein 1Zfp-42, mRNA-seq, determination, Rex1, REX-1, chemical analysis, assay, Zinc finger protein 754, mREX-1, Rex-1, DEL., hREX-1, Reduced expression protein 1falsemRNA-seq analysis of rex-1 deletion in C. elegansmRNA-seq analysis of rex-1 deletion in C. elegans2025-09-242024-05-07PRJNA1105103GSE2659896239