{"database":"ENA","file_versions":[],"scores":null,"additional":{"omics_type":["Genomics"],"center_name":["Max Delbrueck Center Berlin"],"full_dataset_link":["https://www.ebi.ac.uk/ena/browser/view/PRJNA1285140"],"long_description":["The goal of the study is to establish Prime Editing (PE) in differentiated, non-dividing human cardiomyocytes to precisely repair patient-derived DCM mutations to wildtype. We focus on three pathogenic DCM-causing mutations within the human LMNA and RBM20 genes. Our results support further pre-clinical development of PE-based therapies, particularly for RBM20-P633L-associated DCM"],"repository":["ENA"],"additional_accession":[]},"is_claimable":false,"name":"","description":"Prime Editing Corrects RBM20-Mediated Dilated Cardiomyopathy in Human Cardiomyocytes","dates":{"last_updated":"2025-07-04","first_public":"2025-07-04"},"accession":"PRJNA1285140","cross_references":{}}