<HashMap><database>ENA</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR866/SRR866222/SRR866222.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR866/SRR866221/SRR866221.fastq.gz</Fastqsanger.gz></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><center_name>Deepak Srivastava, Gladstone Institute of Cardiovascular Disease, J. David Gladstone Institutes</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA204502</full_dataset_link><scientific_name>Homo sapiens</scientific_name><long_description>We are interested in the role of NOTCH1 and Shear Stress in Aortic Valve Endothelium. Overall design: Primary aortic valve endothelial cells were infected with NOTCH1 intracellular domain fused with a myc tag in order to perform ChIP-seq.</long_description><repository>ENA</repository></additional><is_claimable>false</is_claimable><name>Homo sapiens</name><description>Shear Stress and NOTCH1 Regulate Calcification Related Genes in Human Aortic Valve Endothelium (ChIP-Seq)</description><dates><last_updated>2025-09-24</last_updated><first_public>2015-05-20</first_public></dates><accession>PRJNA204502</accession><cross_references><GEO>GSE47152</GEO><taxon>9606</taxon></cross_references></HashMap>