{"database":"ENA","file_versions":[],"scores":null,"additional":{"omics_type":["Genomics"],"center_name":["Duke University"],"full_dataset_link":["https://www.ebi.ac.uk/ena/browser/view/PRJNA255417"],"scientific_name":["Homo sapiens"],"long_description":["MiR-33a is involved in the maintenance of Glioma Initiating Cells (GIC) and tumor progression. MicroRNA-33a could promote GIC growth and self-renewal by regulating two pathways including cAMP/PKA pathway and Notch pathway. We used microarrays to identify the direct target genes of miR-33a in a glioblastoma cell line D456MG. We used microarrays to detail the global change of gene expression after miR-33a over-expression and identified target genes during this process. Overall design: Cells were infected with lenti-virus expressing a control vector (pWPXLD) or human primary miR-33a. Then RNA was extracted and gene expression was profiled by Affymetrix microarray."],"tag":["xref:PubMed:25202981"],"repository":["ENA"],"additional_accession":[]},"is_claimable":false,"name":"Homo sapiens","description":"Expression data for miR-33a over-expression in CD133-negative D456MG cells","dates":{"last_updated":"2025-09-24","first_public":"2014-07-18"},"accession":"PRJNA255417","cross_references":{"GEO":["GSE59484"],"taxon":["9606"],"PubMed":["25202981"]}}