ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR190/008/SRR1909148/SRR1909148.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR190/007/SRR1909147/SRR1909147.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR190/006/SRR1909146/SRR1909146.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR190/009/SRR1909149/SRR1909149.fastq.gzprimaryOK200GenomicsDepartment of Biology, Johns Hopkins Universityhttps://www.ebi.ac.uk/ena/browser/view/PRJNA277877Caenorhabditis elegansxref:PubMed:26669440In this study we show that global levels of mature miRNAs are unaffected in C. elegans after knockdown of either subunit of CK2 (encoded by kin-3 and kin-10) by RNAi. Overall design: Small RNAs were quantified from wild-type animals at L4 stage fed either vector (L4440), kin-3, kin-10, or alg-1 RNAi. Mature miRNA counts were quantified by summing the total number of reads mapping to each miRNA and normalized to the total number of mapped reads per sequencing library.ENAfalseCaenorhabditis elegansCasein kinase II promotes target silencing by miRISC through direct phosphorylation of the DEAD-box RNA helicase CGH-12025-09-242016-10-10PRJNA277877GSE66764623926669440