<HashMap><database>ENA</database><scores/><additional><omics_type>Genomics</omics_type><center_name>Ontario Institute for Cancer Research</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA285705</full_dataset_link><scientific_name>Homo sapiens</scientific_name><long_description>Goal of the study was to determine if replication stress associated with reprogramming of somatic cells into iPS cells contributes to somatic mutations.</long_description><repository>ENA</repository><description_synonyms>INO1, ichthyosis-prematurity syndrome, human being, Man (Taxonomy), IPS, Modern, ino1-B, Exomes, isyna1, ichthyosis congenita IV, IPS-1, human, Human, FATP4, Homo sapiens, INOS, Modern Man, ips, ichthyosis congenita 4, ACSVL4, ino1-a, ino1, IPS 1, Nucleotide, inos, whole exome., Man, nucleotides</description_synonyms><name_synonyms>Human, Modern., human being, Man (Taxonomy), Homo sapiens, man, Man, human, Modern Man</name_synonyms></additional><is_claimable>false</is_claimable><name>Homo sapiens</name><description>nucleotide supplemented human iPS exome</description><dates><last_updated>2025-09-24</last_updated><first_public>2015-06-05</first_public></dates><accession>PRJNA285705</accession><cross_references><taxon>9606</taxon></cross_references></HashMap>