ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/001/SRR5663681/SRR5663681.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/008/SRR5663688/SRR5663688.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/004/SRR5663684/SRR5663684.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/000/SRR5663680/SRR5663680.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/005/SRR5663685/SRR5663685.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/006/SRR5663686/SRR5663686.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/003/SRR5663683/SRR5663683.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/007/SRR5663687/SRR5663687.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR566/002/SRR5663682/SRR5663682.fastq.gzprimaryOK200GenomicsPing Xu, Philips Institute for Oral Health Research, Virginia Commonwealth Univ. School of Dentistryhttps://www.ebi.ac.uk/ena/browser/view/PRJNA389801Streptococcus sanguinis SK36Gene expression profile of the Streptococcus sanguinis two component system regulator, ciaR, deletion mutant compared to the wild-type strain SK36 Overall design: Experiment: ciaR mutant vs. wild-type SK36 and ciaR complementary strain vs. wild-type SK36. Biological replicates independently grown and prepared for each sample.pathogen:bacteriumxref:PubMed:29215019pathogenbacteriaENAfalseStreptococcus sanguinis SK36Analysis of differential gene expression in the Streptococcus sanguinis ciaR mutant2025-09-242018-03-01PRJNA389801GSE9986438891929215019