<HashMap><database>ENA</database><scores/><additional><omics_type>Genomics</omics_type><center_name>Department of Molecular Oncology, Chiba University</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA400046</full_dataset_link><scientific_name>Homo sapiens</scientific_name><long_description>To confirm the differential regulation could occur in gastric epithelial cells as well as fibroblasts, RNA-seq analysis was performed in GES1 cells treated by pLKO lentiviral vectors expressing shRNA against non-target (shNON), TP53, and TGFBR2. Overall design: Immortalized non-neoplastic gastric epithelial cells, GES1, were transfected with pLKO lentiviral vectors against non-target (shNON), TP53, and TGFBR2. GES1 cells were exposed to puromycin for 4 days for selection, and RNA was extracted. RNA-seq analysis was performed using NexSeq.</long_description><repository>ENA</repository></additional><is_claimable>false</is_claimable><name>Homo sapiens</name><description>Genetic and epigenetic gene inactivation coupling with KRAS activation in human gastric cancer [RNA-Seq]</description><dates><last_updated>2025-09-24</last_updated><first_public>2025-08-16</first_public></dates><accession>PRJNA400046</accession><cross_references><GEO>GSE103045</GEO><taxon>9606</taxon></cross_references></HashMap>