<HashMap><database>ENA</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/006/SRR7169736/SRR7169736_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/004/SRR7169734/SRR7169734_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/003/SRR7169733/SRR7169733_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/007/SRR7169737/SRR7169737_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/004/SRR7169734/SRR7169734_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/005/SRR7169735/SRR7169735_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/005/SRR7169735/SRR7169735_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/006/SRR7169736/SRR7169736_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/003/SRR7169733/SRR7169733_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR716/007/SRR7169737/SRR7169737_2.fastq.gz</Fastqsanger.gz></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><center_name>Shanghai Institute of Immunology</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA471628</full_dataset_link><long_description>Single NG2-glia with GFP fluorescence labeling from PDGFRαCreER mGFP mice was selected and aspirated into a glass pipette from hippocampal acute slices. In brief, cells were picked promptly by micromanipulation and immediately placed in lysis buffer. All NG2 glial cells were collected within 3 h after slice preparation. The selected NG2-glia were processed for single-cell RNA extraction and reverse transcription within 1 h and were subjected to RNA-sequencing.</long_description><repository>ENA</repository></additional><is_claimable>false</is_claimable><name></name><description>Kir4.1 channels in NG2-glia play a role in development, potassium signaling, and ischemia-related myelin loss.</description><dates><last_updated>2023-05-17</last_updated><first_public>2018-05-19</first_public></dates><accession>PRJNA471628</accession><cross_references/></HashMap>