<HashMap><database>ENA</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/005/SRR7592285/SRR7592285_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/004/SRR7592284/SRR7592284_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/001/SRR7592281/SRR7592281_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/002/SRR7592282/SRR7592282_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/006/SRR7592286/SRR7592286_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/004/SRR7592284/SRR7592284_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/001/SRR7592281/SRR7592281_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/005/SRR7592285/SRR7592285_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/003/SRR7592283/SRR7592283_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/003/SRR7592283/SRR7592283_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/002/SRR7592282/SRR7592282_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR759/006/SRR7592286/SRR7592286_1.fastq.gz</Fastqsanger.gz></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><center_name>Deepak Nihalani, Medicine, Medical University of South Carolina</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA482859</full_dataset_link><scientific_name>Homo sapiens</scientific_name><tag>xref:PubMed:30709661</tag><long_description>In this study we plan to compare the profiles of control sample (C) with the disease (FSGS) samples to identify differentially expressed genes. We hope to identify genes that are specifically activated in response to treatment with FSGS plasma. Overall design: Upregulated genes on incubating with plasma from recurrent FSGS plamsa sample in cultured human podocytes cells were probed</long_description><repository>ENA</repository></additional><is_claimable>false</is_claimable><name>Development of a novel cell-based assay to diagnose recurrent Focal and Segmental Glomerulosclerosis</name><description>Development of a novel cell-based assay to diagnose recurrent Focal and Segmental Glomerulosclerosis</description><dates><last_updated>2025-09-22</last_updated><first_public>2018-09-03</first_public></dates><accession>PRJNA482859</accession><cross_references><GEO>GSE117669</GEO><taxon>9606</taxon><PubMed>30709661</PubMed></cross_references></HashMap>