ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR123/087/SRR12375087/SRR12375087.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR123/090/SRR12375090/SRR12375090.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR123/086/SRR12375086/SRR12375086.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR123/091/SRR12375091/SRR12375091.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR123/088/SRR12375088/SRR12375088.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR123/089/SRR12375089/SRR12375089.fastq.gzprimaryOK200GenomicsMarkus Muschen, Center of Molecular and Cellular Oncology, Yale Universityhttps://www.ebi.ac.uk/ena/browser/view/PRJNA650503Mus musculusTo elucidate the dependence of B-ALL cells on PTEN we analysed the transcriptional changes upon inducible Pten KO in BCR-ABL1 transformed pre-B cells. Overall design: Transciptome analysis of Pten+/+ and Pten-/- cells transduced with BCR-ABL1 oncogene.xref:PubMed:33149299ENAfalseRole of PTEN in B-ALLRole of PTEN in B-ALL2025-09-242020-10-21PRJNA650503GSE1556181009033149299