<HashMap><database>ENA</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/070/SRR13294970/SRR13294970_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/072/SRR13294972/SRR13294972_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/073/SRR13294973/SRR13294973_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/069/SRR13294969/SRR13294969_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/068/SRR13294968/SRR13294968_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/073/SRR13294973/SRR13294973_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/071/SRR13294971/SRR13294971_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/070/SRR13294970/SRR13294970_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/071/SRR13294971/SRR13294971_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/072/SRR13294972/SRR13294972_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/068/SRR13294968/SRR13294968_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR132/069/SRR13294969/SRR13294969_1.fastq.gz</Fastqsanger.gz></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><center_name>Dermatology, Stanford University</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA687497</full_dataset_link><scientific_name>Homo sapiens</scientific_name><tag>xref:PubMed:35585237</tag><long_description>The goal of these ATAC-seq experiments was to determine changes in chromatin accesiblity caused by loss of Gibbin (AHDC1) or GATA3 in non-neural ectoderm. Overall design: There were six ATAC-seq samples total including WT, GKO, and GATA3 KO. All samples were perfoemd in biological duplicate.</long_description><repository>ENA</repository></additional><is_claimable>false</is_claimable><name>Gibbin drives epithelial development by toggling CTCF binding and DNA methylation</name><description>Gibbin drives epithelial development by toggling CTCF binding and DNA methylation</description><dates><last_updated>2025-09-24</last_updated><first_public>2022-02-15</first_public></dates><accession>PRJNA687497</accession><cross_references><GEO>GSE163769</GEO><taxon>9606</taxon><PubMed>35585237</PubMed></cross_references></HashMap>