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deValue":200,"statusCode":"OK"}],"scores":null,"additional":{"omics_type":["Genomics"],"center_name":["TransImm, U932 - Immunity and Cancer, Institut Curie"],"full_dataset_link":["https://www.ebi.ac.uk/ena/browser/view/PRJNA793840"],"long_description":["Macrophage infiltration is a hallmark of solid cancers and overall macrophage infiltration correlates with lower patient survival and resistance to therapy. Tumor- associated macrophages, however, are phenotypically and functionally heterogeneous. Specific subsets of tumor-associated macrophage might be endowed with antagonistic roles on cancer progression and on the development of anti-tumor immunity. Here, we identify a discrete population of FOLR2 + tissue-resident macrophages in healthy mammary gland and breast cancer primary tumors. FOLR2 + macrophages localize in perivascular areas in the tumor stroma, where they interact with CD8 + T cells. Moreover, FOLR2 + macrophages efficiently prime effector CD8 + T cells ex vivo . The density of FOLR2 + macrophages in tumors positively correlates with better patient survival. This study highlights antagonistic roles for tumor-associated macrophage subsets and paves the way for subset-specific therapeutic interventions in macrophages-based cancer therapies. Overall design: Tumor infiltrating myeloid cells were isolated from human and murine breast tumors for single cell RNA-seq or bulk RNA-seq analysis"],"repository":["ENA"],"additional_accession":[]},"is_claimable":false,"name":"Tissue-resident FOLR2+ macrophages associate with tumor-infiltrating CD8+ T cells and with increased survival of breast cancer patients","description":"Tissue-resident FOLR2+ macrophages associate with tumor-infiltrating CD8+ T cells and with increased survival of breast cancer patients","dates":{"last_updated":"2023-05-19","first_public":"2022-01-07"},"accession":"PRJNA793840","cross_references":{"GEO":["GSE192935"]}}