<HashMap><database>ENA</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/008/SRR18839708/SRR18839708_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/006/SRR18839706/SRR18839706_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/009/SRR18839709/SRR18839709_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/004/SRR18839704/SRR18839704_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/006/SRR18839706/SRR18839706_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/005/SRR18839705/SRR18839705_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/009/SRR18839709/SRR18839709_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/007/SRR18839707/SRR18839707_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/004/SRR18839704/SRR18839704_1.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/005/SRR18839705/SRR18839705_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/008/SRR18839708/SRR18839708_2.fastq.gz</Fastqsanger.gz><Fastqsanger.gz>ftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR188/007/SRR18839707/SRR18839707_1.fastq.gz</Fastqsanger.gz></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Genomics</omics_type><center_name>Beijing Hospital</center_name><full_dataset_link>https://www.ebi.ac.uk/ena/browser/view/PRJNA828578</full_dataset_link><scientific_name>Rattus norvegicus</scientific_name><long_description>The rat tendon injury models were established and divided into three groups: normal control group, injury model group, and celecoxib + lactoferrin treatment group. Then, RNA sequencing and differential expression analysis were performed for samples from injury model group and celecoxib + lactoferrin treatment group on day 14. Next, autophagy/hypoxia/ferroptosis/pyroptosis-related genes retrieved from the corresponding databases and related literatures were downloaded to obtain the genes associated with autophagy/hypoxia/ferroptosis/pyroptosis. Subsequently, functional annotation, protein-protein interaction (PPI) network and transcriptional regulatory network construction for these genes were performed. Overall design: The rat tendon injury models were established and divided into injury model group and celecoxib + lactoferrin treatment group.RNA sequencing and differential expression analysis were performed for samples from injury model group and celecoxib + lactoferrin treatment group on day 14</long_description><repository>ENA</repository></additional><is_claimable>false</is_claimable><name>Screening for crucial biomarkers of tendon injury and repair in a rat model</name><description>Screening for crucial biomarkers of tendon injury and repair in a rat model</description><dates><last_updated>2025-09-24</last_updated><first_public>2022-11-24</first_public></dates><accession>PRJNA828578</accession><cross_references><GEO>GSE201161</GEO><taxon>10116</taxon></cross_references></HashMap>