ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR193/042/SRR19309642/SRR19309642_2.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR193/041/SRR19309641/SRR19309641_2.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR193/040/SRR19309640/SRR19309640_2.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR193/041/SRR19309641/SRR19309641_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR193/042/SRR19309642/SRR19309642_1.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR193/040/SRR19309640/SRR19309640_1.fastq.gzprimaryOK200GenomicsDepartment of Life Science, Hanyang Universityhttps://www.ebi.ac.uk/ena/browser/view/PRJNA839640Homo sapiensTo understand the pathogenic mechanisms of bone erosion in rheumatoid arthritis (RA), we investigated osteoclast-specific epigenetic programs, RANKL-responsive super-enhancers (SEs) and SE-associated enhancer RNAs (SE-eRNAs) in human osteoclasts. In this dataset, we sought for functional SE-eRNAs in RA patients' synovial monocytes. Overall design: RNA_MCSF and RANKLENAfalseTranscriptome Analysis of Synovial CD14+ cells in Rheumatoid Arthritis PatientsTranscriptome Analysis of Synovial CD14+ cells in Rheumatoid Arthritis Patients2025-09-242022-11-24PRJNA839640GSE2033579606