ENAapplication/xmlftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR247/019/SRR24724619/SRR24724619.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR247/024/SRR24724624/SRR24724624.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR247/023/SRR24724623/SRR24724623.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR247/020/SRR24724620/SRR24724620.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR247/022/SRR24724622/SRR24724622.fastq.gzftp://ftp.sra.ebi.ac.uk/vol1/fastq/SRR247/021/SRR24724621/SRR24724621.fastq.gzprimaryOK200GenomicsAir Force Medical Centerhttps://www.ebi.ac.uk/ena/browser/view/PRJNA974959In this study, we sought to explore the potential ligand-receptor crosstalk andintracellular mechanisms underlying PD-L1-mediated ECs hyperpermeability inhuman lung microvascular endothelial cells (HMVECs).ENAfalsePD-L1/CD80 regulates pulmonary endothelial barrier functions through mtROS/NLRP3/caspase-1 mediated pyroptotic pathway2023-06-032023-06-03PRJNA974959