Ontology highlight
ABSTRACT:
SUBMITTER: Ding,Ma
REPOSITORIES: NODE
Jiang Yi-Zhou YZ Ma Ding D Suo Chen C Shi Jinxiu J Xue Mengzhu M Hu Xin X Xiao Yi Y Yu Ke-Da KD Liu Yi-Rong YR Liu Yi-Rong YR Yu Ying Y Zheng Yuanting Y Li Xiangnan X Zhang Chenhui C Hu Pengchen P Zhang Jing J Hua Qi Q Zhang Jiyang J Hou Wanwan W Ren Luyao L Bao Ding D Li Bingying B Yang Jingcheng J Yao Ling L Zuo Wen-Jia WJ Zhao Shen S Gong Yue Y Ren Yi-Xing YX Zhao Ya-Xin YX Yang Yun-Song YS Niu Zhenmin Z Cao Zhi-Gang ZG Stover Daniel G DG Verschraegen Claire C Kaklamani Virginia V Daemen Anneleen A Benson John R JR Takabe Kazuaki K Bai Fan F Li Da-Qiang DQ Wang Peng P Shi Leming L Huang Wei W Shao Zhi-Ming ZM
Cancer cell 20190307 3
We comprehensively analyzed clinical, genomic, and transcriptomic data of a cohort of 465 primary triple-negative breast cancer (TNBC). PIK3CA mutations and copy-number gains of chromosome 22q11 were more frequent in our Chinese cohort than in The Cancer Genome Atlas. We classified TNBCs into four transcriptome-based subtypes: (1) luminal androgen receptor (LAR), (2) immunomodulatory, (3) basal-like immune-suppressed, and (4) mesenchymal-like. Putative therapeutic targets or biomarkers were iden ...[more]