Ontology highlight
ABSTRACT:
OTHER RELATED OMICS DATASETS IN: PRJNA92717
ORGANISM(S): Homo sapiens
SUBMITTER: Cynthia L Innes William K Kaufmann Stella O Sieber Pierre R Bushel Alexandra N Heinloth Kristina G Flores Paula B Deming Richard S Paules Cynthia Innes
PROVIDER: E-GEOD-3174 | ArrayExpress | 2010-07-01
SECONDARY ACCESSION(S): GSE3174PRJNA92717
REPOSITORIES: GEO, ArrayExpress
Action | DRS | |||
---|---|---|---|---|
E-GEOD-3174.README.txt | Txt | |||
E-GEOD-3174.idf.txt | Idf | |||
E-GEOD-3174.processed.1.zip | Processed | |||
E-GEOD-3174.sdrf.txt | Txt |
Items per page: 5 1 - 4 of 4 |
Innes Cynthia L CL Heinloth Alexandra N AN Flores Kristina G KG Sieber Stella O SO Deming Paula B PB Bushel Pierre R PR Kaufmann William K WK Paules Richard S RS
Molecular cancer research : MCR 20060301 3
The heritable disorder ataxia telangiectasia (AT) is caused by mutations in the AT-mutated (ATM) gene with manifestations that include predisposition to lymphoproliferative cancers and hypersensitivity to ionizing radiation (IR). We investigated gene expression changes in response to IR in human lymphoblasts and fibroblasts from seven normal and seven AT-affected individuals. Both cell types displayed ATM-dependent gene expression changes after IR, with some responses shared and some responses v ...[more]